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Even before scientists sort this out, this information is actually really good news for us,
即使科學家還尚未解決問題，這一好消息確實振奮人心，
because it turns out that the way we live can influence the accumulation of amyloid plaques.
因為這證明了我們的生活方式可以影響淀粉樣斑塊的積累。
And so there are things we can do to keep us from reaching that tipping point.
我們可以做許多小事來避免達到臨界點。
Let's picture your risk of Alzheimer's as a see-saw scale.
不妨把患老年癡呆的風險比作天平。
We're going to pile risk factors on one arm, and when that arm hits the floor, you are symptomatic and diagnosed with Alzheimer's.
把可能增加風險的因素放在一端，如果一端觸到地面，那么你將患病。并被診斷為老年癡呆。
Let's imagine you're 50 years old.
假設你已五十歲。
You're not a spring chicken anymore, so you've accumulated some amyloid plaques with age. Your scale is tipped a little bit.
你已不再年輕，隨年歲增長你已積累了部分淀粉樣斑塊。你的天平已經微微傾斜。
Now let's look at your DNA. We've all inherited our genes from our moms and our dads.
現在觀察一下你的DNA。我們的基因全部遺傳自我們的父母。
Some of these genes will increase our risk and some will decrease it.
一些基因會增加風險，還有一些會降低風險。
If you're like Alice in "Still Alice," you've inherited a rare genetic mutation that cranks out amyloid beta,
如果你像《依然愛麗絲》中的愛麗絲一樣，你遺傳的一種罕見的基因突變，粗制濫造著β淀粉樣蛋白，
and this alone will tip your scale arm to the ground.
單單這一點就會使你的天平一端著地。
But for most of us, the genes we inherit will only tip the arm a bit.
不過對于大多數人而言，該基因只會使天平稍稍傾斜。
For example, APOE4 is a gene variant that increases amyloid,
舉個例子，APOE4是一種增加淀粉樣蛋白的變異基因，
but you can inherit a copy of APOE4 from mom and dad and still never get Alzheimer's,
你可能會從父母那里遺傳一份APOE4的基因，不過卻不會患上老年癡呆，
which means that for most of us, our DNA alone does not determine whether we get Alzheimer's.
這也就意味著對大多數人而言，我們的DNA并不是決定老年癡呆的唯一要素。
So what does? We can't do anything about getting older or the genes we've inherited.
那么什么決定呢？我們對于衰老無能為力，也無法決定我們遺傳的基因。
So far, we haven't changed our brain's destiny.
到現在我們還沒有改變我們大腦的宿命。
