評論
打印
So as you would expect and at anytime you do a stress study like this you would expect a linear increase.
大家可以想象不管什么時候 當你做一個這樣的壓力研究時你都可以看到
In these pro-inflammatory cytokines as what you get traditionally And that's definitely what we've found.
這些前炎性細胞因子呈線性增加的趨勢這和傳統結論是一致的我們的結論也是如此
So this isn't looking at any other factors this is just on average individuals showed an increase in IL-6 in response to the acute stressor.
我們沒有考慮其他因素一般人只是顯示出在面對短期壓力時IL-6水平的上升
And the very same thing is seen for the IL-6 to IL-10 This is significant time effect Then we found that poor sleep.
而且IL-6和IL-10的比例也出現了相同的變化這是一個重要的時間效應然后我們發現睡眠不好的情況
But not BMI these were looked at separately potentiated IL-6.
沒有考慮身體質量指數這些因素都是分別考察的在面對短期壓力時 睡眠不好會影響到
And IL-6/IL-10 responses to acute stress So here you see this So you get this kind of graded effect.
IL-6水平和IL-6和IL-10的比例大家看看這張圖這里有一個梯度效應
That good sleepers had the least slope Average sleepers had a steeper increase but the poor sleepers had a very strong high increase.
睡眠良好的人坡度最緩睡眠一般的人坡度稍陡而睡眠不好的人坡度上升最快
Suggesting that poor sleep is in some way operating to moderate the effect of this stressor on biology specifically in this case it's IL-6/IL-10. But it's the same for IL-6.
這說明了 睡眠不好以某種方式調節了壓力對生物機能的影響特別是IL-6和IL-10的比例, 但對IL-6的影響也是一樣的.
But interestingly it was the combination between BMI and sleep.
然而有趣的是只有當BMI和睡眠結合起來時
That was the worst So here we see poor sleepers with low BMI had the weak slope.
才會出現最糟情況這里大家可以看到BMI指數低睡眠質量不好的人坡度較緩
The good sleepers and this is... Is broken down by BMI because the interaction between good sleepers and BMI was non-significant.
而睡眠質量好的人… BMI在這里發生了失效因為如果睡眠質量好和BMI一般那么它們之間的關系并不顯著
